optic neuromyelitis pedigree disorder (NMOSD) is an autoimmune inflammatory demyelinating disease of the central nervous system. About half of the patients have optic neuritis (ON) as the initial symptom. Long segment spinal cord inflammation (myelitis), severe optic neuritis and / or intractable vomiting and hiccups (postpartum regional syndrome) are the main manifestations of the disease, which can eventually lead to serious sequelae such as permanent blindness, paralysis, and even death. Optic neuritis caused by optic neuromyelitis spectrum disorder is a high-risk disease that seriously affects the vision of patients in neuroophthalmology. With the further study of the pathogenesis of NMOSD, this paper puts forward and analyzes the clinical manifestation of optic neuritis associated with optic neuromyelitis spectrum disease (NMOSD-ON), and puts forward many treatments for its different stages. This article reviews the pathogenesis, clinical characteristics and treatment of optic neuritis associated with optic neuromyelitis spectrum disease.
optic neuromyelitis pedigree disorder (NMOSD) is an autoimmune inflammatory demyelinating disease of the central nervous system. About half of the patients have optic neuritis (ON) as the initial symptom. Long segment spinal cord inflammation (myelitis), severe optic neuritis and / or intractable vomiting and hiccups (postpartum regional syndrome) are the main manifestations of the disease, which can eventually lead to serious sequelae such as permanent blindness, paralysis, and even death. Optic neuritis caused by optic neuromyelitis spectrum disorder is a high-risk disease that seriously affects the vision of patients in neuroophthalmology. With the further study of the pathogenesis of NMOSD, this paper puts forward and analyzes the clinical manifestation of optic neuritis associated with optic neuromyelitis spectrum disease (NMOSD-ON), and puts forward many treatments for its different stages. This article reviews the pathogenesis, clinical characteristics and treatment of optic neuritis associated with optic neuromyelitis spectrum disease.
Objective: To investigate whether the early stage hordeolum is treated with hot compress or cold compress. Methods: From March 2019 to March 2021, 129 patients (129 eyes) who were diagnosed with early hordeolum in our outpatient department were randomly divided into two groups according to the order of visits. They were treated with cold compress or hot compress, 3 times a day. Times, 10-15 minutes each time. And with tobramycin eye drops, oral administration of roxithromycin and other drugs. The observation time is 1-3 days. Results: Of the 65 cases in the hot compress group, 15 cases were effective and 40 cases were ineffective. Among them, 35 cases gave up hot compress treatment, with a total effective rate of 23.08%. Of the 64 cases in the cold compress group, 20 cases were cured, 44 cases were effective, and the total effective rate was 100%. Conclusion: Cold compress can relieve symptoms, control inflammation, relieve pain, and improve the comfort of the affected area. It is the best choice for early meibinitis.
摘要 目的 探究原发性急性闭角型青光眼与脉络膜厚度、眼轴长度及前房深度的相关性。方法 随机选取2020年3月-2022年3月在我院进行诊治的80例（109眼）原发性急性闭角型青光眼患者（急性组）作为研究对象进行回顾性分析研究，选取同时期在本院进行检查的正常房角眼受检者17例（47眼）作为对照组。采用频域光学相干断层扫描增强技术检测脉络膜厚度相关指标。采用人工晶体光学生物测量仪检测眼轴长度变化。采用超声生物显微镜检测前房深度变化。Pearson相关性检验分析脉络膜厚度、眼轴长度及前房深度间的相关关系。结果 与对照组对比，急性组患者受检眼球中心凹颞侧2500μm（T2.5）、眼球中心凹鼻颞侧2500μm（N2.5）及黄斑中心凹下脉络膜厚度（SFCT）水平均明显降低（P<0.05）。与对照组对比，急性组患者眼轴长度、前房深度及前房深度/眼轴长度水平均明显降低（P<0.05）。经Pearson相关性检验，T2.5、SF脉络膜厚度、N2.5、眼轴长度、前房深度及前房深度/眼轴长度间均存在显著正相关（P<0.05）。结论 原发性急性闭角型青光眼患者具有脉络膜薄、眼轴长度短、前房深度浅的特点，且脉络膜厚度与眼轴长度、前房深度具有明显正相关关系。
